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Erratum to: Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases

  • Eri Fukuda1, 2,
  • Katarzyna H Kaminska4, 3,
  • Janusz M Bujnicki1, 4, 3 and
  • Ichizo Kobayashi1, 2, 5Email author
Genome Biology201212:412

https://doi.org/10.1186/gb-2011-12-11-412

Received: 16 November 2011

Accepted: 23 November 2011

Published: 15 January 2012

The original article was published in Genome Biology 2008 9:R163

Correction

We have noticed an error in the sixth paragraph of the Discussion of our article [1]. The corrected paragraph should read:

Another type IV nuclease, GmrSD, found in an E. coli strain targets glucosylated hydroxymethyl-C and may have evolved to cut T4 genome [78]. The resistance of hydroxymethyl-C-containing phage to restriction enzymes but its sensitivity to McrBC [79], the resistance of glucosylated hydroxymethyl-C to McrBC but sensitivity to GmrSD [78], and inhibition of GmrSD by T4-coded internal protein [78] suggest an evolutionary arms race (evolutionary struggle between competing sets of co-evolving genes that develop adaptations and counter-adaptations against each other) between the bacteria and the phage.

Notes

Authors’ Affiliations

(1)
Laboratory of Social Genome Sciences, Department of Medical Genome Sciences, University of Tokyo, Minato-ku, Japan
(2)
Graduate Program in Biophysics and Biochemistry, Graduate School of Science, University of Tokyo, Minato-ku, Japan
(3)
International Institute of Molecular and Cell Biology, Warsaw, Poland
(4)
Faculty of Biology, Adam Mickiewicz University, Poznan, Poland
(5)
Institute of Medical Science, University of Tokyo, Minato-ku, Japan

References

  1. Fukuda E, Kaminska KH, Bujnicki JM, Kobayashi I: Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases. Genome Biology. 2008, 9: R163-10.1186/gb-2008-9-11-r163.PubMedPubMed CentralView ArticleGoogle Scholar

Copyright

© BioMed Central Ltd. 2012

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