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Table 1 Comparison of key anthropometric data for those with and without hepatic steatosis as measured by ultrasound in the Fenland cohort. A Student’s t-test was used to compare parameters between the two groups

From: Hepatic steatosis risk is partly driven by increased de novo lipogenesis following carbohydrate consumption

  Non-steatosis Steatosis
Individuals assessed by ultrasound (n) 663 233
Women (n (%)) 424 (64.0) 110 (47.2)
Age (years) 45 ± 7 48 ± 7***
BMI (kg/m2) 25.5 ± 3.7 30.7 ± 4.8***
Fat mass (kg) 24.2 ± 7.5 33.8 ± 9.5***
Liver score 3.66 ± 0.47 6.06 ± 1.20***
Fasting plasma insulin (pmol/L) 37.9 ± 23.0 68.0 ± 49.4***
Fasting plasma NEFAs (μM) 355 ± 179 369 ± 181
Alcohol consumption (g/d) 9.4 ± 12.3 10.5 ± 14.1
Total carbohydrate consumption 243 ± 106 237 ± 89
Fasting blood glucose (mM) 4.78 ± 0.59 5.12 ± 0.74***
Fasting blood triglyceride (mM) 1.03 ± 0.65 1.70 ± 0.64***
HOMA-IR 0.80 ± 0.49 1.45 ± 1.04***
Fatty Liver Index 31.1 ± 25.4 70.0 ± 24.4***
  1. ***p < 0.001