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Table 1 Comparison of key anthropometric data for those with and without hepatic steatosis as measured by ultrasound in the Fenland cohort. A Student’s t-test was used to compare parameters between the two groups

From: Hepatic steatosis risk is partly driven by increased de novo lipogenesis following carbohydrate consumption

 

Non-steatosis

Steatosis

Individuals assessed by ultrasound (n)

663

233

Women (n (%))

424 (64.0)

110 (47.2)

Age (years)

45 ± 7

48 ± 7***

BMI (kg/m2)

25.5 ± 3.7

30.7 ± 4.8***

Fat mass (kg)

24.2 ± 7.5

33.8 ± 9.5***

Liver score

3.66 ± 0.47

6.06 ± 1.20***

Fasting plasma insulin (pmol/L)

37.9 ± 23.0

68.0 ± 49.4***

Fasting plasma NEFAs (μM)

355 ± 179

369 ± 181

Alcohol consumption (g/d)

9.4 ± 12.3

10.5 ± 14.1

Total carbohydrate consumption

243 ± 106

237 ± 89

Fasting blood glucose (mM)

4.78 ± 0.59

5.12 ± 0.74***

Fasting blood triglyceride (mM)

1.03 ± 0.65

1.70 ± 0.64***

HOMA-IR

0.80 ± 0.49

1.45 ± 1.04***

Fatty Liver Index

31.1 ± 25.4

70.0 ± 24.4***

  1. ***p < 0.001