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Table 1 Table 1

From: Wrangling for microRNAs provokes much crosstalk

Publication Findings Cellular context
Tay et al. [2] Computational predictions combined with experimental analyses reveal protein-coding ceRNAs that act as trans-regulators of PTEN with similar tumor-suppressive properties Human prostate and colon cancer cell lines
Karreth et al. [3] Transposon insertion within ZEB2 creates a new ceRNA pairing that decreases PTEN expression levels via crosstalk involving miR-181, miR-200b, miR-25 and miR-92a Human melanoma cell lines and a mouse model of melanoma
Cesana et al. [4] Linc-MD1, a muscle-specific long noncoding RNA, regulates muscle differentiation by acting as a ceRNA that competes with MAML1 and MEF2C, transcripts encoding essential transcription factors involved in myogenic differentiation, for miR-133 and miR-135 Various mouse and human muscle cell types
Sumazin et al. [5] Systematic analysis of genome-wide microRNA expression profiles reveals a ceRNA network that regulates key drivers of gliomagenesis through distinct oncogenic pathways and that determines tumor subtype formation Human glioblastoma cell lines