Open Access

Erratum to: Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases

  • Eri Fukuda1, 2,
  • Katarzyna H Kaminska4, 3,
  • Janusz M Bujnicki1, 4, 3 and
  • Ichizo Kobayashi1, 2, 5Email author
Genome Biology201212:412

DOI: 10.1186/gb-2011-12-11-412

Received: 16 November 2011

Accepted: 23 November 2011

Published: 15 January 2012

The original article was published in Genome Biology 2008 9:R163

Correction

We have noticed an error in the sixth paragraph of the Discussion of our article [1]. The corrected paragraph should read:

Another type IV nuclease, GmrSD, found in an E. coli strain targets glucosylated hydroxymethyl-C and may have evolved to cut T4 genome [78]. The resistance of hydroxymethyl-C-containing phage to restriction enzymes but its sensitivity to McrBC [79], the resistance of glucosylated hydroxymethyl-C to McrBC but sensitivity to GmrSD [78], and inhibition of GmrSD by T4-coded internal protein [78] suggest an evolutionary arms race (evolutionary struggle between competing sets of co-evolving genes that develop adaptations and counter-adaptations against each other) between the bacteria and the phage.

Notes

Authors’ Affiliations

(1)
Laboratory of Social Genome Sciences, Department of Medical Genome Sciences, University of Tokyo
(2)
Graduate Program in Biophysics and Biochemistry, Graduate School of Science, University of Tokyo
(3)
International Institute of Molecular and Cell Biology
(4)
Faculty of Biology, Adam Mickiewicz University
(5)
Institute of Medical Science, University of Tokyo

References

  1. Fukuda E, Kaminska KH, Bujnicki JM, Kobayashi I: Cell death upon epigenetic genome methylation: a novel function of methyl-specific deoxyribonucleases. Genome Biology. 2008, 9: R163-10.1186/gb-2008-9-11-r163.PubMedPubMed CentralView Article

Copyright

© BioMed Central Ltd. 2012

Advertisement